Mice deficient for the stable tubule only peptide (STOP) display altered dopaminergic neurotransmission associated with severe behavioural defects including disorganized locomotor activity. Endogenous morphine, which is present in nervous tissues and synthesized from dopamine, may contribute to these behavioral alterations since it is thought to play a role in normal and pathological neurotransmission.

Charlet, A., Muller, A.H., Laux, A., Kemmel, V., Schweitzer, A., Deloulme, J.C., et al. (2010). Abnormal nociception and opiate sensitivity of STOP null mice exhibiting elevated levels of the endogenous alkaloid morphine. MOLECULAR PAIN, 6(1), 96 [10.1186/1744-8069-6-96].

Abnormal nociception and opiate sensitivity of STOP null mice exhibiting elevated levels of the endogenous alkaloid morphine

BIANCHI, ENRICA;
2010-01-01

Abstract

Mice deficient for the stable tubule only peptide (STOP) display altered dopaminergic neurotransmission associated with severe behavioural defects including disorganized locomotor activity. Endogenous morphine, which is present in nervous tissues and synthesized from dopamine, may contribute to these behavioral alterations since it is thought to play a role in normal and pathological neurotransmission.
2010
Charlet, A., Muller, A.H., Laux, A., Kemmel, V., Schweitzer, A., Deloulme, J.C., et al. (2010). Abnormal nociception and opiate sensitivity of STOP null mice exhibiting elevated levels of the endogenous alkaloid morphine. MOLECULAR PAIN, 6(1), 96 [10.1186/1744-8069-6-96].
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11365/976134
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