It is now well established that hyperglycemia, present in both type 1 and type 2 diabetes, causes a variety of biochemical derangements leading to a diffused vascular damage responsible for several pathologic manifestations. Although preclinical and clinical studies have been performed by an unreliable administration route, the correct approach of oxygen-ozonetherapy may break a vicious circle. Messengers, released by a precise interaction ex vivo of the patient’s blood with an equivalent calculated dose of ozone (0.42–0.84 mM), react with a variety of cells after blood infusion and restore a number of functions went astray. This paper aims to open a debate on this new therapy for improving the prognosis of diabetes.

Bocci, V., Zanardi, I., Huijberts, M.S.P., Travagli, V. (2011). Diabetes and chronic oxidative stress. A perspective based on the possible usefulness of ozone therapy. DIABETES & METABOLIC SYNDROME, 5, 45-49 [10.1016/j.dsx.2010.05.014].

Diabetes and chronic oxidative stress. A perspective based on the possible usefulness of ozone therapy

BOCCI, VELIO;ZANARDI, IACOPO;TRAVAGLI, VALTER
2011-01-01

Abstract

It is now well established that hyperglycemia, present in both type 1 and type 2 diabetes, causes a variety of biochemical derangements leading to a diffused vascular damage responsible for several pathologic manifestations. Although preclinical and clinical studies have been performed by an unreliable administration route, the correct approach of oxygen-ozonetherapy may break a vicious circle. Messengers, released by a precise interaction ex vivo of the patient’s blood with an equivalent calculated dose of ozone (0.42–0.84 mM), react with a variety of cells after blood infusion and restore a number of functions went astray. This paper aims to open a debate on this new therapy for improving the prognosis of diabetes.
2011
Bocci, V., Zanardi, I., Huijberts, M.S.P., Travagli, V. (2011). Diabetes and chronic oxidative stress. A perspective based on the possible usefulness of ozone therapy. DIABETES & METABOLIC SYNDROME, 5, 45-49 [10.1016/j.dsx.2010.05.014].
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11365/8299
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