Host specific T-cell response is critical for the outcome of Helicobacter pylori infection. In genetically susceptible individuals, H. pylori can activate gastric CD4(+) Th1 cells that recognize cross-reactive epitopes shared by H. pylori proteins and self H+, K+-ATPase, leading to gastric autoimmunity via molecular mimicry. (C) 2004 Elsevier SAS. All rights reserved.
D'Elios, M.M., Bergman, M.P., Amedei, A., Appelmelk, B.J., Del Prete, G. (2004). Helicobacter pylori and gastric autoimmunity. MICROBES AND INFECTION, 6(15), 1395-1401 [10.1016/j.micinf.2004.10.001].
Helicobacter pylori and gastric autoimmunity
D'Elios, Mario Milco;
2004-01-01
Abstract
Host specific T-cell response is critical for the outcome of Helicobacter pylori infection. In genetically susceptible individuals, H. pylori can activate gastric CD4(+) Th1 cells that recognize cross-reactive epitopes shared by H. pylori proteins and self H+, K+-ATPase, leading to gastric autoimmunity via molecular mimicry. (C) 2004 Elsevier SAS. All rights reserved.
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